EGCG Rescues deficits in DS Mouse model and in human beings full text PDF

This section contains both peer reviewed materials a articles written with the layperson in mind. Prefrontal Cortex Drives Distinct Projection Neurons in the Basolateral Amygdala Aliminum In the Brain of Autistic Children – full text Curcumin and Omega 3 fatty acids in treatment of Alzheimer’s disease in patients with and without Down Syndrome – the importance of the pleiotropic Intervention (TNI is a pleiotropic intervention). Aging in DS and the development of Alzheimer’s disease Differential Expression of Inflammatory Genes in DS. SLCP (LongVida) Curcumin why it’s best in the treatment of gene over expression INFLUENCE OF NUTRIENTS ON REGULATORY T CELLS ALUMINUM IS THE PRIMARY ETIOLOGICAL FACTOR IN ALL FORMS OF ALZHEIMERS DSEASE COCONUT OIL AND ALZHEIMERS DISEASE DEKITERIOUS EFFECT OF CHRIST NIC FOLATE DEFICIENCY IN DS WHAT IS DHA AND WHAT DOES IT DO? INHIBITORY EFFECTS OF RESVERATROL ON EVB VITAMIN C PREVENTS PROLIFERATION OF EBV TREHALOSE DESOLVES AD/DS AMALOID PROTEIN PLAQUE  TREHALOSE AND AUTISM TCTP AND THE PANCREAS TCTP AND INSULIN RESISTANCE  METHYLTETRAHYDRFOLATE DEFICIENCY AND INFANTILE SEIZURES  BETA GLUCAN AND AUTO IMMUNE DISEASE SOLID TUMOR MALIGNANCIES ARE RARE IN CHILDREN WITH DS EFFECTS OF FAUTLY METHYLATION ASTROGLIAL CELLS AND METABOLISM AMYLOID PROTEIN TOXICITY AND CANABINOIDS HMGN1 AND LEUKEMIA IN DS ROLE OF GLUTATHIONE NASAL SPRAY IN EAR INFECTION NEBULIZED GLUTATHIONE NOT RECOMMENDED STUDY SHOWS UNVACCINATED CHILDREN ARE HEALTHIER ACETAMINOPHEN AND AUTISM Cerebral Spinal Fluid Markers in DS The burden of additional genes in DS disrupts the proteostasis network. Normalizing DS biochemistry by down regulating excess genes may prevent this. Micronutrient profiles in patients with DS. Note this study does not necessarily conform to what we see in untreated individuals with DS. Review of AD biomarkers in DS The DS Brain in the presence or absence of neuro fibrillation. TNI addresses this issue by down regulating the genes involved and assisting the glymphatic system in removing Amyloid protein particles. Repetitive Behaviors in DS Diabetes in DS is not associated with a congenital deficiency in B-cells. Brain predictive age and cognitive decline – Amyloid protein related. Again, this is not an issue in treated children. Screening for Celiac Disease in DS Comparison of balance and gait in DS and neuro typical children.