IMG_0140

EGCG Rescues deficits in DS Mouse model and in human beings full text PDF

This section contains both peer reviewed materials a articles written with the layperson in mind.

Prefrontal Cortex Drives Distinct Projection Neurons in the Basolateral Amygdala

Aliminum In the Brain of Autistic Children – full text

Curcumin and Omega 3 fatty acids in treatment of Alzheimer’s disease in patients with and without Down Syndrome – the importance of the pleiotropic Intervention (TNI is a pleiotropic intervention).

Aging in DS and the development of Alzheimer’s disease

Differential Expression of Inflammatory Genes in DS.

SLCP (LongVida) Curcumin why it’s best in the treatment of gene over expression

INFLUENCE OF NUTRIENTS ON REGULATORY T CELLS

ALUMINUM IS THE PRIMARY ETIOLOGICAL FACTOR IN ALL FORMS OF ALZHEIMERS DSEASE

COCONUT OIL AND ALZHEIMERS DISEASE

DEKITERIOUS EFFECT OF CHRIST NIC FOLATE DEFICIENCY IN DS

WHAT IS DHA AND WHAT DOES IT DO?

INHIBITORY EFFECTS OF RESVERATROL ON EVB

VITAMIN C PREVENTS PROLIFERATION OF EBV

TREHALOSE DESOLVES AD/DS AMALOID PROTEIN PLAQUE 

TREHALOSE AND AUTISM

TCTP AND THE PANCREAS

TCTP AND INSULIN RESISTANCE 

METHYLTETRAHYDRFOLATE DEFICIENCY AND INFANTILE SEIZURES 

BETA GLUCAN AND AUTO IMMUNE DISEASE

SOLID TUMOR MALIGNANCIES ARE RARE IN CHILDREN WITH DS

EFFECTS OF FAUTLY METHYLATION

ASTROGLIAL CELLS AND METABOLISM

AMYLOID PROTEIN TOXICITY AND CANABINOIDS

HMGN1 AND LEUKEMIA IN DS

ROLE OF GLUTATHIONE NASAL SPRAY IN EAR INFECTION

NEBULIZED GLUTATHIONE NOT RECOMMENDED

STUDY SHOWS UNVACCINATED CHILDREN ARE HEALTHIER

ACETAMINOPHEN AND AUTISM

Cerebral Spinal Fluid Markers in DS

The burden of additional genes in DS disrupts the proteostasis network. Normalizing DS biochemistry by down regulating excess genes may prevent this.

Micronutrient profiles in patients with DS. Note this study does not necessarily conform to what we see in untreated individuals with DS.

Review of AD biomarkers in DS

The DS Brain in the presence or absence of neuro fibrillation. TNI addresses this issue by down regulating the genes involved and assisting the glymphatic system in removing Amyloid protein particles.

Repetitive Behaviors in DS

Diabetes in DS is not associated with a congenital deficiency in B-cells.

Brain predictive age and cognitive decline – Amyloid protein related. Again, this is not an issue in treated children.

Screening for Celiac Disease in DS

Comparison of balance and gait in DS and neuro typical children.